Original contributionPolypoid dysplasia in Barrett's esophagus: A clinicopathologic, immunohistochemical, and molecular study of five cases
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Cited by (51)
Gastrointestinal dysplasia
2013, PathologyHistopathology of Barrett's esophagus: A review for the practicing gastroenterologist
2010, Techniques in Gastrointestinal EndoscopyCitation Excerpt :Finally, some dysplastic lesions in BE may grow as exophytic, well-circumscribed, sessile or stalked polypoid lesions that, historically, have been mistermed “adenomas” of the esophagus. Thurberg and coworkers studied these lesions, finding that adenoma-like polypoid dysplastic lesions in BE showed a high association with HGD and adenocarcinoma within the polyp and adjacent flat mucosa.76 Thus, in this circumstance, endoscopic polypectomy is inadequate treatment.
Hyperplastic polyp arising in Barrett's esophagus
2010, Gastroenterologie Clinique et BiologiqueDiffuse Esophageal Polyposis: An Uncommon Occurrence
2009, Annals of Thoracic SurgeryCitation Excerpt :Whether or not polyps of the esophagus are one marker in the progression of the dysplasia-to-carcinoma sequence is unknown. Thurberg and colleagues' [5] report on five cases with sessile or pedunculated polypoid lesions of the esophagus associated with Barrett's esophagus revealed adenocarcinoma arising within the polyp in four of the cases, and one case in the adjacent Barrett's esophagus [5]. Given the uncertainty of the degree of association of esophageal polyposis with progression to adenocarcinoma, resection is recommended, particularly in patients with associated Barrett's esophagus.
Polyps of the Esophagus
2009, Surgical Pathology of the GI Tract, Liver, Biliary Tract and PancreasEpithelial Neoplasms of the Esophagus
2009, Surgical Pathology of the GI Tract, Liver, Biliary Tract and Pancreas