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Research ArticleExperimental Studies

Isothiocyanates Induce Cell Cycle Arrest, Apoptosis and Mitochondrial Potential Depolarization in HL-60 and Multidrug-resistant Cell Lines

JANA JAKUBIKOVA, YONGPING BAO and JAN SEDLAK
Anticancer Research September 2005, 25 (5) 3375-3386;
JANA JAKUBIKOVA
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YONGPING BAO
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JAN SEDLAK
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  • For correspondence: exonsedl@savba.sk
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Article Information

vol. 25 no. 5 3375-3386
PubMed 
16101152

Published By 
International Institute of Anticancer Research
Print ISSN 
0250-7005
Online ISSN 
1791-7530
History 
  • Received February 25, 2005
  • Received May 30, 2005
  • Published online September 1, 2005.

Copyright & Usage 
Copyright© 2005 International Institute of Anticancer Research (Dr. John G. Delinassios), All rights reserved

Author Information

  1. JANA JAKUBIKOVA1,2,
  2. YONGPING BAO1,* and
  3. JAN SEDLAK2
  1. 1Nutrition Division, Institute of Food Research, Norwich Research Park, Norwich, NR4 7UA, U.K.
  2. 2Laboratory of Tumor Immunology, Cancer Research Institute, Vlarska 7, Bratislava, Slovak Republic
  1. Correspondence to: Jan Sedlak, Cancer Research Institute, Vlarska 7, 833 91 Bratislava, Slovak Republic. e-mail: exonsedl{at}savba.sk

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Anticancer Research: 25 (5)
Anticancer Research
Vol. 25, Issue 5
1 Sep 2005
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Isothiocyanates Induce Cell Cycle Arrest, Apoptosis and Mitochondrial Potential Depolarization in HL-60 and Multidrug-resistant Cell Lines
JANA JAKUBIKOVA, YONGPING BAO, JAN SEDLAK
Anticancer Research Sep 2005, 25 (5) 3375-3386;

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Isothiocyanates Induce Cell Cycle Arrest, Apoptosis and Mitochondrial Potential Depolarization in HL-60 and Multidrug-resistant Cell Lines
JANA JAKUBIKOVA, YONGPING BAO, JAN SEDLAK
Anticancer Research Sep 2005, 25 (5) 3375-3386;
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